Your personalised DNA insights, distilled into clear priorities and practical next steps.
We interpret your DNA through a clinician-informed lens to highlight what matters most for your health. Evidence-based, context-aware, and easy to act on, so you can make confident decisions with clarity and calm.
Focused on insights most relevant to your health and life stage.
Curated with robust evidence and genomic science.
Clear practical guidance you can discuss with your clinician.
Transparent about what we report and what we intentionally exclude.
Grouped by what to actually do, with the findings that support each step underneath.
Use one routine sweep: HbA1c or fasting glucose, BP trend, ApoB-inclusive lipids, Lp(a), and waist/body-composition trend.
Keep the foundations steady: sleep consistency, social connection, stress management, regular movement, and avoiding heavy cannabis or stimulant misuse.
Keep a short GI symptom and food-pattern note, alongside family history and red flags. Use clinical tests when symptoms or history make them relevant.
Start with the ordinary reversible causes: Full Blood Count (FBC), thyroid, ferritin, B12/folate, and sleep/recovery context before attributing fatigue to genetics.
Check eGFR, urine ACR/UACR, blood pressure, and glucose context. Those measured results decide whether the genetic signal matters.
A note on what this is. Genetics is useful when it moves the right ordinary check up the list. It does not diagnose conditions or replace measured results, symptoms, family history, or clinician judgement.
Pick the body system you care about first. Each card shows where the report has top actions, routine checks, and background context.
These are the findings that earned a place in the plan. Skim them for the logic; the full library sits underneath.
Your genetics lean toward higher LDL cholesterol. That nudges a routine check forward; the measured result is what decides whether anything needs doing.
Linked to: Lock in a cardiometabolic baselineYour polygenic loading for Type 2 diabetes sits clearly above the population median. It nudges a routine check forward; it does not say you have the condition.
Linked to: Lock in a cardiometabolic baselineYour genome points toward higher HbA1c, which is the blood-sugar side of cardiometabolic health. That nudges a routine glucose check forward; it does not predict diabetes, and measured HbA1c or fasting glucose settles the question.
Linked to: Lock in a cardiometabolic baselineYour genetics lean toward higher ApoB. That nudges a routine check forward; the measured result is what decides whether anything needs doing.
Linked to: Lock in a cardiometabolic baselineYour genetics lean toward higher triglycerides. That nudges a routine check forward; the measured result is what decides whether anything needs doing.
Linked to: Lock in a cardiometabolic baseline
Your polygenic loading for Malaise and fatigue sits clearly above the population median. It nudges a routine check forward; it does not say you have the condition.
Linked to: Lock in a cardiometabolic baseline
Your genetics lean toward higher urinary albumin-to-creatinine ratio. That nudges a routine check forward; the measured result is what decides whether anything needs doing.
Linked to: Lock in a cardiometabolic baselineWeak evidence toward Pi*S/Pi*Z alpha-1 antitrypsin variants (SERPINA1)
Linked to: Lock in a cardiometabolic baseline
Your polygenic loading for Irritable bowel syndrome sits clearly above the population median. It nudges a routine check forward; it does not say you have the condition.
Linked to: Track the gut pattern before testingYour polygenic loading for Ulcerative colitis sits clearly above the population median. It nudges a routine check forward; it does not say you have the condition.
Linked to: Track the gut pattern before testingYour polygenic loading for Inflammatory bowel disease sits clearly above the population median. It nudges a routine check forward; it does not say you have the condition.
Linked to: Track the gut pattern before testing
Your polygenic loading for Anxiety disorder sits clearly above the population median. It nudges a routine check forward; it does not say you have the condition.
Linked to: Keep mental-health foundations steadyYour polygenic loading for Attention-deficit/hyperactivity disorder sits clearly above the population median. It nudges a routine check forward; it does not say you have the condition.
Linked to: Keep mental-health foundations steadyYour polygenic loading for Depression sits clearly above the population median. It nudges a routine check forward; it does not say you have the condition.
Linked to: Keep mental-health foundations steady
Your polygenic loading for Primary open-angle glaucoma sits clearly above the population median. It nudges a routine check forward; it does not say you have the condition.
Linked to: Lock in a cardiometabolic baselineYour polygenic loading for Acne sits clearly above the population median. It nudges a routine check forward; it does not say you have the condition.
Linked to: Lock in a cardiometabolic baseline| Area | Priority | What to check | Why now | Constraints |
|---|---|---|---|---|
| Lock in a cardiometabolic baseline | High | Use one routine sweep: HbA1c or fasting glucose, BP trend, ApoB-inclusive lipids, Lp(a), and waist/body-composition trend. |
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Do not infer current diabetes, hypertension, lipid disease, or medication need; measured results decide action. |
| Keep mental-health foundations steady | Medium | Keep the foundations steady: sleep consistency, social connection, stress management, regular movement, and avoiding heavy cannabis or stimulant misuse. |
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Do not infer a psychiatric diagnosis or treatment need from PRS alone; symptoms and functional change decide concern. |
| Track the gut pattern before testing | Medium | Keep a short GI symptom and food-pattern note, alongside family history and red flags. Use clinical tests when symptoms or history make them relevant. |
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Do not start restrictive diets or infer IBD/celiac disease from PRS alone. |
| Rule out reversible fatigue causes | High | Start with the ordinary reversible causes: Full Blood Count (FBC), thyroid, ferritin, B12/folate, and sleep/recovery context before attributing fatigue to genetics. |
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Do not infer a fatigue diagnosis; common reversible causes and lived symptoms matter more than PRS. |
| Confirm kidney and urine markers | High | Check eGFR, urine ACR/UACR, blood pressure, and glucose context. Those measured results decide whether the genetic signal matters. |
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Do not infer kidney disease from PRS alone; repeat measured kidney and urine results decide follow-up. |
| PGx medication fileVariants affecting response to drugs | Conditional | Record these medication contexts in the medical records:
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Not actionable without a prescribing event. |
